Associate Professor within the Faculty of Microbiology – Masters Program in Bioinformatics and Systems Biology, University of Costa Rica
Rodrigo Mora conducted his PhD studies in the field of Programmed Cell Death in Cancer at the German Cancer Research Center (DKFZ, Heidelberg), and his first postdoctoral training in Systems Biology at the Bioquant Center at the University of Heidelberg. Currently, he holds the position of Associate Professor within the Faculty of Microbiology, and the Masters Program in Bioinformatics and Systems Biology at the University of Costa Rica. Over the past five years, he has successfully established his own research group, the Systems Biology of PersonalizedTherapy, and has been responsible for supervising several PhD student, Masters students, and undergraduate students both in biological and computational fields. The University of Costa Rica is very interested to create a Program of Translational Medicine due to high availability of tumor samples in the country, which could allow him, in the future, to set up collaborations with his connections in Germany. However, it is still very hard for scientists on the fields of Cancer and Systems Biology, as this is considered basic science. International collaboration is therefore essential to succeed. His research interests include the Systems Biology of personalised cancer treatment. His research team is currently performing an in vitro test of chemosensitivity on tumors of breast cancer patients. The remaining tumoral material can be used to perfom sequencing studies looking for genomic/transcriptomic markers of resistance/sensitivity to chemotherapy and other basic research projects on the understanding of cancer biology, specially the role of miRNA and lncRNAs in cancer stability. In addition, his team is also working on the development of molecular sensors for several applications: i. detect chemotherapy resistance at the single cell level, based on sphingolipid metabolism and autophagy, ii. The study of Dengue virus pathogenesis and Dengue virus-induced cellular alterations such as autophagy and cell death mechanisms.